Overcoming GLP-1 Resistance Through Cortisol Modulation to Unlock More Effective Weight Loss in Dys-Metabolic Obesity
9:30 am - Thursday 10th December 2026
- Exploring how chronic stress, HPA-axis dysregulation, and cortisol excess contribute to treatment-resistant obesity, diabetes, and hypertension
- Examining the biological mechanisms linking cortisol signalling with appetite regulation, energy balance, and responsiveness to incretin-based therapies
- Assessing emerging therapeutic strategies targeting cortisol synthesis, metabolism, and action to enhance weight loss, improve metabolic health, and preserve muscle mass